CARM1 and BAF155: an example of how chromatin remodeling factors can be relocalized and contribute to cancer

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dc.contributor.author	Stefansson, Olafur A.
dc.contributor.author	Esteller, Manel
dc.date	2018-11-12T09:34:52Z
dc.date	2018-11-12T09:34:52Z
dc.date	2014-05-19
dc.date	2018-11-12T09:34:52Z
dc.identifier.citation	1465-542X
dc.identifier.citation	662732
dc.identifier.uri	http://hdl.handle.net/2445/126004
dc.format	application/pdf
dc.language.iso	eng
dc.publisher	BioMed Central
dc.relation	Reproducció del document publicat a: https://doi.org/10.1186/bcr3657
dc.relation	Breast Cancer Research, 2014, vol. 16, num. 3, p. 307
dc.relation	https://doi.org/10.1186/bcr3657
dc.rights	cc-by (c) Stefansson, Olafur A. et al., 2014
dc.rights	info:eu-repo/semantics/openAccess
dc.rights	http://creativecommons.org/licenses/by/3.0/es
dc.subject	Càncer
dc.subject	Metàstasi
dc.subject	Cromatina
dc.subject	Cancer
dc.subject	Metastasis
dc.subject	Chromatin
dc.title	CARM1 and BAF155: an example of how chromatin remodeling factors can be relocalized and contribute to cancer
dc.type	info:eu-repo/semantics/article
dc.type	info:eu-repo/semantics/publishedVersion
dc.description.abstract	In a recent article, Wang and colleagues reported the discovery of a mechanism by which CARM1 regulates the genomic localization of BAF155 (a SWI/SNF subunit involved in chromatin remodeling) through post-translational methylation at R1064 arginine residues. This modification leads to the relocalization of BAF155-containing SWI/SNF complexes to regions containing genes involved in the Myc oncogenic pathway. The results presented are evidence that these interactions constitute a mechanism by which the BAF155 chromatin remodeling factor contributes to cancer.

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