To access the full text documents, please follow this link: http://hdl.handle.net/10230/41854
dc.contributor.author | Hipótilo, Lucia |
---|---|
dc.contributor.author | Fakira, Amanda K. |
dc.contributor.author | Cabañero Ferri, David |
dc.contributor.author | Blandón, Rebecca |
dc.contributor.author | Carlton, Susan M. |
dc.contributor.author | Morón, Jose A. |
dc.contributor.author | Melyan, Zara |
dc.date | 2015 |
dc.identifier.citation | Hipólito L, Fakira AK, Cabañero D, Blandón R, Carlton SM, Morón JA et al. In vivo activation of the SK channel in the spinal cord reduces the NMDA receptor antagonist dose needed to produce antinociception in an inflammatory pain model. Pain. 2015 May; 156(5): 849-58. DOI: 10.1097/j.pain.0000000000000124 |
dc.identifier.citation | 0304-3959 |
dc.identifier.citation | http://dx.doi.org/10.1097/j.pain.0000000000000124 |
dc.identifier.uri | http://hdl.handle.net/10230/41854 |
dc.format | application/pdf |
dc.language.iso | eng |
dc.publisher | Lippincott Williams & Wilkins |
dc.rights | info:eu-repo/semantics/openAccess |
dc.rights | © Lippincott Williams & Wilkins "This is a non-final version of an article published in final form in Hipólito L, Fakira AK, Cabañero D, Blandón R, Carlton SM, Morón JA et al. In vivo activation of the SK channel in the spinal cord reduces the NMDA receptor antagonist dose needed to produce antinociception in an inflammatory pain model. Pain. 2015 May; 156(5): 849-58. http://dx.doi.org/10.1097/j.pain.0000000000000124 |
dc.subject | Indole |
dc.subject | Inflammation |
dc.subject | Oxima |
dc.subject | Dolor -- Tractament |
dc.subject | Medul·la espinal |
dc.title | In vivo activation of the SK channel in the spinal cord reduces the NMDA receptor antagonist dose needed to produce antinociception in an inflammatory pain model |
dc.type | info:eu-repo/semantics/article |
dc.type | info:eu-repo/semantics/acceptedVersion |
dc.description.abstract | |
dc.description.abstract |