TP53INP2 at the crossroad of apoptosis and autophagy in death receptor signaling

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dc.contributor	Universitat de Barcelona
dc.contributor.author	Ivanova, Saska
dc.contributor.author	Zorzano Olarte, Antonio
dc.date	2019-12-18T14:28:01Z
dc.date	2019-06-24
dc.date	2019-12-18T14:02:15Z
dc.date	info:eu-repo/date/embargoEnd/2020-06-24
dc.identifier.uri	http://hdl.handle.net/2445/146977
dc.format	3 p.
dc.format	application/pdf
dc.language.iso	eng
dc.publisher	Taylor and Francis
dc.relation	Versió postprint del document publicat a: http://dx.doi.org/10.1080/23723556.2019.1632687
dc.relation	Molecular & Cellular Oncology, 2019, vol. 6, num. 5, p. e1632687
dc.relation	http://dx.doi.org/10.1080/23723556.2019.1632687
dc.rights	(c) Taylor and Francis, 2019
dc.rights	info:eu-repo/semantics/embargoedAccess
dc.subject	Autofàgia
dc.subject	Marcadors bioquímics
dc.subject	Autophagy
dc.subject	Biochemical markers
dc.title	TP53INP2 at the crossroad of apoptosis and autophagy in death receptor signaling
dc.type	info:eu-repo/semantics/article
dc.type	info:eu-repo/semantics/acceptedVersion
dc.description.abstract	The binding of ligands to death receptors elicits distinct outcomes, such as apoptosis, inflammation and necroptosis, depending on the cellular context. We have recently described that the autophagic protein TP53INP2 favors apoptosis upon death receptor signaling and is a potential biomarker of responsiveness to TRAIL treatment.